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Agonists targeting α 2 -adrenergic receptors (α 2 AR) [133], opioid receptors [134], or chemokine receptors [135] normally activate ERK through Gβγ–Ras or Gβγ–PI3K signaling modules
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This then causes inflammation in the body as it begins to attack the intruders, and eventually leads to autoimmunity
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