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An example of a GSH analogue was obtained through the chemical modification of γ-L-glutamyl-L-cysteinylglycine (GSH) into γ-glutamyl-S-(benzyl)cysteinyl-phenylglycine diethyl ester (i.e., ezatiostat or TLK199) that is easily absorbed by the cell where its metabolites bind the G-site (the GSH binding site) of GSTP1-1 causing its inhibition [111]

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We take the same position on other compounds at this stage of development, which is why our review of why 5-amino-1MQ has zero registered human trials despite being marketed aggressively for fat loss, and what the preclinical NNMT research actually demonstrated reaches the same conclusion about a different molecule