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It counteracted free radical formation regardless of the effect on NO, whether an increase or decrease [80,144,175,181,182,192,193,194,195,196,197]

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However, under oxidative or chemical stress conditions, GSTP1-1 dissociates from the complex and accumulates in oligomeric structures, resulting in the release and activation of JNK for subsequent phosphorylation of downstream targets regulating cell proliferation and apoptosis [60]