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Ondansetron is also a weak antagonist of the 5-HT1B, 5-HT1C, alpha-adrenergic, and opioid mu receptors
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However, during oxidative stress (e.g., due to excessive ROS), Nrf2 is released and translocates to the nucleus, where it promotes the expression of antioxidant defence genes, such as glutathione reductase and superoxide dismutase [17]
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