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Furthermore, the oxidative damage in the SN of PD patients contributes to mtDNA damage as has been seen in FRDA ( These hallmarks of disease pathology in PD mouse SN neurons were suppressed with CSA treatment (Stack et al., 2008)

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ROS production triggers the activation of mitogen-activated protein kinase (MAPK) signaling and the redox-sensitive transcription factor NF-κB (nuclear factor kappa light chain enhancer of activated B cells) [11], which orchestrates the transcription of pro-inflammatory genes

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