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This may result in altered neuronal signaling and circuit-wide plasticity deficiencies that could contribute to the cognitive and other symptoms associated with schizophrenia and neuropsychiatric illnesses (Fig

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After confirming the concentrations and times of administration of CS and GHK-Cu to cells, we observed that RAW264.7 cells and primary AMs produce more ROS due to an excessive phagocytosis of CS (50 μg/cm 2 ), whereas GHK-Cu reversed this phenomenon (Fig