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cAMP signaling of adenylate cyclase toxin blocks the oxidative burst of neutrophils through Epac-mediated inhibition of phospholipase C activity
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Mitochondrial dysfunction in ME/CFS Mitochondrial dysfunction is implicated as a key pathogenic process in ME/CFS, underpinned by mechanisms involving oxidative and nitrosative stress, immune-inflammatory pathways, and metabolic disturbances
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