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Combined mineralocorticoid and glucocorticoid deficiency is caused by a novel founder nicotinamide nucleotide transhydrogenase mutation that alters mitochondrial morphology and increases oxidative stress
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Fingolimod, a drug which first arose due its role on lymphocyte migration, showed the ability to induce angiogenesis by inducing a M2 phenotype in microglia through S1PR1 modulation (158) and STAT3 signaling (159)
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