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doi: 10.1016/j.freeradbiomed.2024.10.296 174 LiYXuWZhaoGGuoYWangLDuQet al

Allergy Research Group

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In the context of human-like hydrophobic BA models, the activation of the cytokine-c-Jun N-terminal kinases (JNKs)-mitogen-activated protein kinases (MAPKs) signaling pathway by the FXR receptor emerges as a pivotal mechanism for regulating BA synthesis during acute liver injury, such as that induced by cholesterol [40]