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Additionally, the administration of curcumin has demonstrated therapeutic potential by negatively regulating various signaling pathways (PI3/Akt, JAK/STAT, and β-catenin), inhibiting the expression of COX-2, iNOS, and co-stimulatory molecules (RANKL, ICAM-1, CD205, CD256, TLR4, among others), and suppressing immune cell chemotaxis, including the infiltration of neutrophils into inflamed regions (Zhao et al., 2016
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