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Our conceptual reconstruction [4,5,6,7] took Robert’s prototypical model of intragastric alcohol-induced gastric lesions [1,2,3] as its starting-point
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In level 1 analyses, contrasts were modeled to examine the brain reactivity to correctly-responded Go trials and correctly-inhibited NoGo trials in the Go/NoGo task and inhibitory control in the Stop-signal task
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